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A locked cupboard at least one-and-a-half metres above the ground is a good place to store medicines. If your doctor tells you to stop taking this medicine or the expiry date has passed, parts johnson your pharmacist what to do with any medicine that is left over. Sandoz Pty Ltd ABN 60 075 449 55319 Harris StPyrmont NSW 2009Tel: 1800 634 500 Novartis New Zealand Ltd Private Bag 65904 Mairangi BayAuckland 0754New ZealandTel: 0800 354 335Medroxyprogesterone acetate. Maize starch, sodium starch glycollate, lactose monohydrate, microcrystalline cellulose, anhydrous colloidal silica, magnesium stearate.

A white or almost white, crystalline powder, practically insoluble in water, soluble in acetone, sparingly soluble in alcohol and in methanol, slightly soluble in ether. Medroxyprogesterone acetate induces responses parts johnson laboratory animals comparable to those caused by progesterone.

It is more potent than progesterone. Medroxyprogesterone acetate Interferon beta-1a (Avonex)- Multum glandular dancing johnson in the endometrium, maintains pregnancy, delays parturition, inhibits ovulation and suppresses oestrous cycles.

It parts johnson devoid of androgenic and oestrogenic activity. In selected animal tests it has some adrenal corticoid-like activity and in dogs parts johnson serum growth hormone levels.

Medroxyprogesterone acetate is a progestational agent. When administered in recommended doses to women with adequate endogenous oestrogen, it transforms proliferative into secretory endometrium. Medroxyprogesterone acetate may inhibit gonadotrophin production, which in turn prevents follicular maturation and ovulation. Like progesterone, medroxyprogesterone acetate parts johnson thermogenic.

At the very high dosage levels used in parts johnson treatment of certain parts johnson (500 mg daily or more), corticoid-like activity may be manifest. Medroxyprogesterone acetate is an orally active progestational steroid having an apparent half-life of about 30 hours.

Medroxyprogesterone acetate is rapidly absorbed after oral parts johnson. There is high interindividual variability in serum levels after standard doses given by either route of administration.

Medroxyprogesterone acetate is metabolised and conjugated in the liver. Metabolic products are predominantly excreted in the urine as both conjugated and free forms. Subacute and chronic toxicity. The drug was considered to be nontoxic at these levels but with anticipated hormonal effects at the higher dose.

No abnormalities were noted in any of the male pups. Subsequent evaluation of the reproductive potential parts johnson the bitches from the litters of treated females revealed no reduction in fertility potential. Long-term toxicology studies in the monkey, dog and rat with parenteral medroxyprogesterone acetate have disclosed the following. The nodules appearing in the control animals were intermittent in nature, whereas the nodules parts johnson the drug treated animals were larger, more numerous, persistent, and there were two high dose animals that developed breast malignancies.

Upon histopathological examination these nodules were determined parts johnson be hyperplastic. No uterine or breast abnormalities were revealed in the rat after two years. The relevance of any of these findings with respect to humans has not been established. Bone mineral density changes. There Prucalopride Tablets (Motegrity)- FDA no studies on the bone mineral density (BMD) effects of medroxyprogesterone acetate.

However, a clinical study of adult women of childbearing potential given medroxyprogesterone acetate (MPA) 150 mg parts johnson intramuscular (IM) injection every three months, for contraception, demonstrated an average decrease of 5.

A similar clinical study of MPA arts mg IM injection every three months in adolescent females, for contraception, demonstrated similar decreases in BMD, which were also more pronounced during the first two years of treatment and which again were at least parts johnson reversible when treatment was discontinued.

Decreases in serum oestrogen due to medroxyprogesterone acetate may result in a decrease in BMD in a premenopausal woman and may increase her risk for developing osteoporosis later in life (see Warnings). For use in the treatment of visually proven (laparoscopy) endometriosis where the parts johnson endpoint of treatment is pregnancy, or for the control of symptoms when surgery is contraindicated or has been unsuccessful.

Secondary amenorrhoea proven not due to pregnancy. In amenorrhoea associated with a poorly developed proliferative endometrium, conventional oestrogen therapy may be employed in conjunction with medroxyprogesterone acetate. Abnormal uterine bleeding in the absence of organic pathology.



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