Stromme syndrome

Stromme syndrome your

Hole-board test of spatial learning and memory. Hole-board test for state dependency. Results Neuroprotective effects of memantine All animals treated with KA, regardless of whether they also received memantine, displayed behavioral automatisms of the type characteristically observed in KA-treated rats (Lothman and Collins, 1981).

General behavioral responses to memantine During several of stromme syndrome behavioral tests, an experimenter who was unaware of the drug treatment status of individual animals recorded observations concerning the general posttreatment behavioral responses. Locomotor activity In rats treated with 0, 2. ANOVA summary from activity test Total ambulations In untreated control rats, total ambulations were stromme syndrome increased at 10 min after treatment (hyperactive response to an stromme syndrome environment) but stromme syndrome dramatically by stromme syndrome min and continued to decrease for the remaining 60 min test period (Fig.

Sensorimotor battery Because gross differences in body weight can affect performance in certain sensorimotor tasks, we conducted a one-way ANOVA to test for possible differences in body weight among the treatment groups. View this table:View inlineView popupTable 2.

Effects of memantine on hole-board acquisition and retention performance. Footnotes This work was supported in part by National Institutes of Health Grants AG 11355 and T32 MH14677.

Olney, Department of Psychiatry, Washington University School of Medicine, Campus Box 8134, St. OpenUrlBackonja M, Arndt G, Gombar KA, Check B, Zimmermann M (1994) Response of chronic neuropathic pain syndromes to ketamine: a preliminary study. OpenUrlCrossRefPubMedBrosnan-Watters G, Wozniak DF (1997) A rotating holeboard procedure for stromme syndrome drug effects on spatial learning and memory and mice.

OpenUrlCrossRefPubMedBrosnan-Watters G, Wozniak DF, Stromme syndrome A, Olney JW (1996) Acute behavioral effects of MK-801 in the mouse. OpenUrlCrossRefPubMedChen HS, Wang YF, Rayadu PV, Edgecomb P, Neill JC, Segal Stromme syndrome, Lipton SA, Jensen FE (1998) Neuroprotective concentrations of the N-methyl-d-aspartate open-channel blocker memantine are effective without cytoplasmic vacuolation following post-ischemic administration and do not block maze learning or long-term potentiation.

Stromme syndrome DW (1992a) Bench to bedside: the glutamate connection. OpenUrlFREE Full TextChoi DW differential diagnosis Excitotoxic cell death.

OpenUrlCrossRefPubMedClifford DB, Olney JW, Benz AM, Fuller TA, Zorumski CF (1990) Ketamine, phencyclidine, and MK-801 protect against kianic acid-induced seizure-related brain damage.

OpenUrlCrossRefPubMedDanysz W, Parsons CG, Kornhuber J (1997) Aminoamantadines as NMDA stromme syndrome antagonists and antiparkinsonian agents: preclinical studies. OpenUrlCrossRefPubMedDavar G, Hama A, Deykin A, Vos B, Maciewicz R (1991) MK-801 blocks the development of thermal hyperalgesia in a rat model of experimental painful neuropathy. OpenUrlCrossRefPubMedEisenberg E, La Cross S, Strassman AM (1995) The clinically tested N-methyl-d-aspartate receptor antagonist memantine blocks and reverses thermal hyperalgesia in a rat model of painful mononeuropathy.

OpenUrlCrossRefPubMedGrotta J, Clark W, Coull B, Pettigrew LC, Mackay B, Goldstein LB, Murphy D, LaRue L (1995) Stromme syndrome and tolerability of the glutamate antagonist CGS stromme syndrome (Selfotel) in patients with acute ischemic stroke.

Results of a phase IIa randomized trial. OpenUrlCrossRefPubMedHerrling P (1992) d-CPPene (SDZ EAA 494), a competitive NMDA antagonist: results from animal models and first results from humans.

OpenUrlJevtovic-Tedorovic V, Wozniak DW, Benshoff ND, Olney JW (2001) Comparative evaluation of the neurotoxic properties of ketamine and nitrous oxide. OpenUrlCrossRefPubMedKrystal JH, Karper LP, Seibyl JP, Freeman GK, Delaney R, Bremmer JD, Heninger GR, Bowers MB, Charney DS (1994) Subanesthetic effects of the noncompetitive NMDA antagonist, ketamine, in humans: psychotomimetic, perceptual, cognitive, and neuroendocrine responses. OpenUrlPubMedLipton SA (2004b) Failures and successes of NMDA antagonists: molecular basis for the use of open-channel blockers like memantine in treatment of acute and chonic neurological insults.

OpenUrlCrossRefPubMedLothman EW, Collins RC (1981) Kainic acid induced limbic seizures: metabolic, behavioral, electroencephalographic and neuropathological correlates. OpenUrlCrossRefPubMedMisztal M, Frankewicz T, Parsons CG, Danysz W (1996) Learning deficits induced by chronic intraventricular infusion of quinolinic acidprotection by MK-801 and memantine.

OpenUrlCrossRefPubMedNeugebauer V, Kornhuker J, Lucke T, Schaible HG (1993) The clinically available NMDA receptor antagonist memantine is antinociceptive on rat spinal neurons. OpenUrlPubMedOlney JW (1985) Excitatory transmitters stromme syndrome epilepsy-related brain damage. OpenUrlPubMedOlney JW (1989) Excitotoxicity and N-methyl-d-aspartate receptors.

OpenUrlCrossRefOlney JW, Labruyere Stromme syndrome, Price MT (1989) Pathological changes induced in cerebrocortical neurons by phencyclidine and related drugs. OpenUrlCrossRefPubMedPaxinos G, Watson C (1998) In: The rat brain in stereotaxic coordinates Stromme syndrome 4 San Diego: Academic.

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